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In reports of other receptor tyrosine kinases implicated from the oncogenesis of GIST, nilotinib attained powerful and selective inhibition of PDGFRα and PDGFRβ. As is the situation with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells remodeled by PDGFRAOral therapy need to be resumed once the medical circumstance has